Sudden Infant Death Syndrome (SIDS) Essay -- Crib Death SIDS

Sudden Infant devastation Syndrome (SIDS)Sudden Infant Death Syndrome (SIDS) or crib death is an abrupt and inexplicable death of an obviously healthy infant. Most of the cases involve infants from ages 1-12 months, and the event occurs during the night. Various theories get to been postulated from look results but without consistency of the etiology. Since the death is sudden, prior diagnostic criteria or patterns be not available for correlation, although some near-miss infants have been followed. A number of possibilities have been documented in current literature, to include beta-endorphin changes, abnormal temperature regulation, pineal abnormalities, carotid body irregularities, lead poisoning, elevated fetal hemoglobin, brainstem immaturity, and cerebral hypoperfusion. The pursuit is an overview of these pathologies in their relation to Sudden Infant Death Syndrome. As with some physiological processes, several intermediate steps can lead to a certain event, thus making the mechanism more controlled. However, as more steps that are required, there arises a greater number of executable problems. SIDS is no exception. Most literature supports the view that victims of SIDS stick a failure of the automatic control of respiration, producing periodic apnea and eventually death. neuronal control of respiration involves three anatomical structures (Armstrong et al., 1982. The first is the motor system, which contains the neurons which start up and maintain respiration. These include the dorsal motor gist of the vague, the nucleus tractus solitarius, the nucleus ambiguous, the nucleus retro-ambiguous, the reticulo-spinal tracts in the anterior and lateral columns and the anterior horn cells of the cervical and thora... ...161122-1126, 1978. Koceard-Varo, G. The physiological role of the pineal gland as the masterswitch of life, turning on at birth breathing and geared to it the function of the autonomic neural system. The cause of SIDS examined in this context. Medical Hypothesis, 34122-126, 1991. Myer, E., Morris, D., et. al. Increased cerebrospinal fluid beta-endorphin immunoreactivity in infants with apnea and in siblings of victims of Sudden Infant Death Syndrome. J. Pedia., 111660-666, 1987. Quattrochi, J., McBride, P., and Yates, A. brainstem immaturity in Sudden Infant Death Syndrome A decimal rapid Golgi study of dendritic spines in 95 infants. Brain Research, 32539-48, 1985. Takashima, S., Armstrong, D., Becker, L., et. al. rational hypoperfusion in the Sudden Infant Death Syndrome? Brainstem gliosis and vasculature. Ann. Neurol., 4257-262, 1978.